infarct
infarct
1. scintigraphic measurement of infarct volume
(心肌)梗塞体积的闪烁照相测量
2. Both MCE and TTC showed that the mass of infarct myocardium of IP group was less than the IR group.
3D MCE及TTC染色结果均显示IP组心肌受损范围小于IR组。
3. The larger the infarct size of CI patient is,the lower the PLT is,the higher the MPV、PDW and Fn is.
4.脑梗塞患者梗塞面积越大，PLT越低，MPV、PDW及血浆Fn越高。
4. CT scan was detected in 50 cases with VD and 141 infarct lesions were discovered (left 79 and right 62).
50例VD患者行CT检查 ,检出141个梗塞灶 ,左侧79个 ,右侧62个。
5. The infarct volume ratios of the rats of group A was tended to be smaller and more inconsistent than group B (coefficient of variation (CV) of the group A is 1.08, CV of the group B is 0.12).
B组大鼠脑梗死体积比明显较A组稳定,变异系数(coefficient of variation, CV )分别为CVB =0.12,CVA=1.08); 再灌注10min和再灌注24h对大鼠进行神经功能缺失评分,B组评分均高于A组(P<0.05);
6. CTA scanning revealed blood vessels convulsion, constriction and obstruction in infarct area.
CTA显示梗塞区血管痉挛、收缩、中断 ,显示出血区造影剂外漏、局部呈无血管区。
7. In focal cerebral ischemia, pretreatment with EDT 2.5, 5, and 10 mg/kg and nimodipine 2 mg/kg for 5 d effectively improved the abnormal neurological symptoms and reduced the infarct rate.
EDT 2.5、5和10mg/kg及尼莫地平2mg/kg灌胃5d显著改善局灶性脑缺血小鼠的神经运动功能,缩小脑梗塞范围。
8. The Application of FLAIR to cerebral infarct
FLAIR成像技术在脑梗塞磁共振诊断中的应用价值
9. There was significant more infarct in the placenta of ICP patients.
ICP患者胎盘发生梗死率明显高于正常妊娠组;
10. The ratio of RCA to the total number of infarct related arteries (IRA) was much higher than that of the left circumflex artery (LCX) (86.1% versus 13.9%).
IRA的分布也是 RCA多于左旋支 (L CX) ,分别占 86 .1%和 13.9%。
11. Infarct size(IR/AAR)in ATV group were reduced(P<0.01).
TSOD升高、MDA降低(P<0.01)。
12. With TTC staining,there were no significent differences among three groups in AAR( P >0 05),NIC group produced a marked reduction in infarct size compared with IR and GLIB+NIC group( P <0 01).
TTC测定 AAR范围 :各组之间无显著性差异 (P>0 .0 5 )。 TTC测定 IA范围 :NIC组较 IR组和 GL IB+ NIC组明显缩小 (P<0 .0 1)。
13. scintigraphic measurement of infarct vol-ume
[心肌]梗死体积的闪烁照相测量
14. Some organs such as bowel with anastomosing blood supplies, or liver with a dual blood suppy, are hard to infarct.
一些器官如肠侧支循环较多，或者如肝有双重血液供应，很难发生梗死。
15. Compared with I/R,the myocardial swelling,interstitial hemorrhage and inflammatory cell infiltrate were decreased,the myocardial infarct size was reduced(P<0.01) after CIP and RIP.
与I/R组比较,CIP和RIP均能明显改善心肌损害的形态学改变,降低心肌细胞的肿胀、减少间质出血和炎性细胞的浸润;
16. Haemorrhagic transformation of infarct and severe adverse events were similar in both groups.
两组中梗塞转化为出血及出现严重不良事件的情况类似。
17. Haemorrhagic transformation of infarct and seere aderse eents were similar in both groups.
两组中梗塞转化为出血及出现严重不良反应（应该是＂事件＂，而不是＂反应＂）的情况类似。
18. Conclusion Nicergoline was an effective and safe drug in treatment of multi infarct dementia.
两组均无明显药物相关的不良反应。
19. Volume of infarct focus of rats in the two groups.
两组大鼠梗死灶体积。
20. Hindman N B, Schocken D D, Windmann M, et al. Evaluation of a QRS scoring system for estimating myocardial infarct size[J]. Am J Cardiol,1985,55:1485.
中华心血管病杂志编委会.急性心肌梗死溶栓疗法参考方案[J].中华心血管病杂志,1991,19:37.
21. The extent of neutrophilic infiltrates, along with karyolysis and loss of cytoplasmic cross striations, is typical for an acute myocardial infarct of 1 to 4 days in duration from onset.
中性粒细胞广泛浸润，核溶解、胞浆减少，是典型的急性心肌梗死的1-4天的表现。
22. It has been reported that MMP-2 and MMP-9 are activated after onset of stroke in clinical practice as well as in experimental brain ischemia and inhibiting the activityof MMP-9 can reduce brain infarct.
临床上已有报道MMP-2及MMP-9的活性在脑中风发作后明显升高。 动物实验也显示局部脑缺血后MMP-9在早期就被激活，抑制MMP-9的活性可以减小脑梗塞面积。
23. The net protective of adenosine in invivo models are the reduction of the infarct size and reduce cardiovascular mortality.
临床研究发现腺苷可以减小心肌梗死范围，降低心血管事件死亡率。
24. To determine the outcome of the biomaterial after injection, calcium-crosslinked biotin-labeled alginate was injected into the infarct 7 days after anterior myocardial infarction in rat.
为了测定注射后生物材料的结果，小鼠前壁心肌梗塞7天后钙离子交联型生物素标记海藻酸钠被注入梗塞部位。
25. MAIN OUTCOME MEASURES: Incidence of animal model and infarct volume.
主要观察指标:动物模型出现率;梗死体积。
26. Subsequent imaging with MR revealed extension of the infarct into the left posterior cerebral artery territory.
之后的磁共振显示梗塞的范围以扩大到左侧大脑后动脉区域。
27. Behavior disorders were observed at 3, 6, 12h after the operation. Willis circle, infarct volume and histopathologic changes were examined 12 hours after operation.
于手术后 3、6、12h观察行为障碍并评分 ,手术后 12h取脑观察Willis环、脑梗死体积及病理改变。
28. Score 1.5 as criterion,incidence of HF and CS in patient whose QRS score increased over 1.5 was higher than those of under 1.5.Conclusion Patient with RI had larger infarct size than patient with IE.
以 QRS记分增加值 1.5分为界 ,与 IE和 RI发生前比较 QRS记分增加值超过 1.5分者心衰及心脏性休克的发生率明显多于未超过 1.5分者。 结论 RI患者的梗死面积大于 IE患者。
29. Cerebral infarct, intracranial hematoma and edema are deadly diseases, which can cause high deformity and death rate.
但是,目前还缺乏一种无创的辅助诊断技术与设备,用于对颅内血肿水肿进行早期、实时的监测。
30. For example, in clinical trials, tirilazad therapy for ischemic stroke increases the risk of death and dependency, whereas in animal studies, it reduces infarct volume and seems to improve neurobehavioral scores.
例如，在临床试验中，替拉扎特治疗缺血性中风增加了其死亡的危险和依赖，尽管在动物实验中他减少了梗塞面积，提高了神经功能评分。
31. All the subjects met the criteria for multi infarct dementia.
入组的病例均符合多发性梗死性痴呆的诊断标准。
32. The mechanisms contributed to ischemic brain injury involve excitotoxicity,calcium overload,free radical reaction,depolarization surrounding infarct,inflammation and apoptosis.
兴奋毒性、钙超载、自由基反应、梗死周围除极、炎症和凋亡是造成缺血性脑损伤的中心环节。
33. One of these unanticipated benefits is improement of the infarct's passie mechanical properties and subsequent amelioration of entricular remodeling (23).
其中一个获益原因可能是梗死区心肌被动机械性能的改善，并由此导致的心室重构的改善。
34. In the reperfusion group, the ratios of the infarct size of the area at risk (IS/ARR) were 45%, 47%and 45%in the early, metaphase and late sham ischemic preconditioning (SIPC) groups respectively.
再灌组,早、晚期假性预适应组坏死质量占缺血质量(IS/AAR)分别高达45%,47%,45%;
35. So apoptosis may contribute to the final infarct size.
凋亡可能决定了最终梗死体积。
36. Hemorrhagic infarction of the primary disease is cerebral infarction, infarct artery after recanalization, but also hemorrhage.
出血性脑梗塞的原发病是脑梗塞，梗塞的动脉血管再通后，又合并出血。
37. Initial work-up included a CT demonstrating a medial left occipital infarct involving the left side of the splenium of the corpus callosum.
初始检查CT示左枕叶及中度梗塞并累及左侧胼胝体压部。
38. Infarct and squamous metaplasia of prostate
前列腺梗死和鳞状细胞化生
39. Here is a cerebral infarct from an arterial embolus, which often leads to a hemorrhagic appearance.
动脉栓子引起的脑梗死,常导致出血性外观。
40. atherosclerotic cerebral infarct in the youth
动脉粥样硬化性脑梗塞
41. The ultrastructure of myocardium cell was slightly destroyed and the infarct size was significantly smaller in the cap-feed group than the normally feed rabbits and CF rabbits.
卡托普利加胆固醇组心肌细胞超微结构结构破坏及心肌梗死面积明显小于对照组和胆固醇组。
42. He was also receiving concomitant carvedilol and an ECG showed only an inferior wall infarct without arrhythmia.
另外，他还同时服用卡维地洛，心电图仅显示下壁心梗，没有心律不齐。
43. The subacute (intermediate) infarct seen here at the right shows edema with obscured structural outlines and swelling that shifts the midline to the left.
右边是亚急性梗死(中速)，出现明显水肿，伴组织结构轮廓不清和肿胀，正中线推向左侧。
44. Right frontal encephalomalacia The frontal pole is largely replaced with the high signal of cerebrospinal fluid left after liquifaction necrosis in this old infarct.
右额叶脑软化额极大部分被旧梗塞灶液化坏死后留下的高信号脑脊液替代。
45. Peripheral facial palsy, deafness, tinnitus, and trigeminal sensory deficit are common presentations.Vertigo as the only clinical feature of AICA infarct is rare.
周边颜面神经麻痹、耳聋、耳鸣及三叉感觉异常为其常见的病徵，但只以晕眩症表现的前下下小脑动脉梗塞更是罕见。
46. Instead, noncontractile fibrous cells gradually replace an infarct's dead myocytes.Healthy myocytes adjacent to the infarct may also die, causing the infarct to expand further.
因此到最后，反而是由不具收缩能力的纤维细胞逐渐取代梗塞坏死的心肌细胞，梗塞区域周围的健康心肌细胞也跟著死亡，导致梗塞区域范围扩大。
47. Thus,DPTI realized a valid method to assess the area of acute infarct myocardium of the left ventricle.
因此可将DPTI作为定量分析急性心肌梗塞面积的可靠方法
48. Figure 1. The correlation between infarct size and risk region of I/R and PC groups in rats.
图1.缺血/再灌注组和缺血预处理组大鼠心肌梗死范围与缺血区域的关系.
49. Note the wedge shape of this zone of coagulative necrosis resulting from loss of blood supply with resultant tissue ischemia that produces the pale infarct.
图示凝固性坏死的区域呈楔形是由于该区域血液供应减少而使组织最终发展为贫血性梗死。
50. The infarction seen here has punctate hemorrhages. This infarct was caused by an embolus.
图示梗死为小斑点状出血，梗死由栓子引起。
51. The infarct also has high signal on the T2- weighted images. This slice also demonstrates the ribbon-like geometry of the lesion.
在T2加权像上，梗塞灶也呈高信号。此层面损害区域同样呈带状。
52. On the next lower slice, extension of the infarct into white matter is seen.
在下一页更低的层面,可以看到延伸至白质的梗塞灶。
53. After controlling for age and gender, the team found associations between a smaller ratio of retinal arterioles and venules with MRI findings, including prevalent infarcts, worsening white matter grade, and incident infarct.
在对年龄和性别进行控制后，研究小组在磁共振结果中发现较小比率视网膜小动脉和小静脉之间的联系，包括普遍的梗塞，恶化的白质程度，以及偶发的梗塞。
54. The fixed tissue piece were aparted under anatomy scope and then weighted. IS,AAR,IS/AAR% and IS/LV% were observed to assess infarct size.
在解剖显微镜下分离IS、AAR ,分别称重 ,观察IS、AAR、IS/AAR %及IS/LV %以评价缺血范围。
55. A bilateral haemorrhagic infarct was observed in affecting the basal ganglia and thalamus.
基底节和丘脑等处可见双侧出血性梗死。
56. Improving the tolerability of mycardium to hypoxia and act against cardiac infarct like propanolol.
增强心肌耐缺氧能力，有与心得安相似的抗心肌梗塞作用。
57. This is an intermediate to remote infarct in the distribution of the middle cerebral artery.
大脑中动脉供血区域的陈旧性梗死。
58. The correlation between anatomical infarction area and NCT image area for infarct focus were r=0.820 for normothermia group (P<0.05) and r= 0.851 for hypothermia group (P<0.05), respectively.
大脑中动脉梗塞模型 ,常温组 ,NCT梗塞面积和染色样本相关系数为 0 .82 0 (P <0 .0 5) ,NCT和fMRI对梗塞灶成像面积的相关系数为 0 .874(P <0 .0 5) ;
59. In the hypothermy environment, obvious increment of human s blood viscosity was observed, which may induce the cerebral infarction, myocardial infarct or coronary heart disease.
太阳活动不仅与人间鼠疫及流行性脑脊髓膜炎两类传染病年发病率间存在着较为密切的相关,而且还与肿瘤的发生和婴儿出生率和死亡率的增加、克山病的发生有关。
60. Keywords Oxiracetam;Cerebral infarct;Sequela;
奥拉西坦;脑梗死;后遗症;
61. If the blood supply is not quickly restored, the bowel will infarct.
如果血液供应没有迅速恢复，肠就会梗死。
62. Kahn JC,Guerel P,Menier R,et al.Prognostic value of enzymatic(CPK) estimation of infarct size.
姚竞.急性心肌梗死早期心电图指标与梗死面积的关系.实用心电学杂志,2002；
63. At the end of reperfusion, ischemic area and infarct size were defined by Evans blue and triphenyltetrazolium chloride staining. The expression of ICAM-1 in myocardium was measuredby RT-PCR.
实验中监测心电图及心功能,实验完毕取心肌组织,Evans蓝及氯化三苯基四氮唑(TTC)染色测心肌梗死程度,逆转录聚合酶链反应法(RT-PCR)测心肌局部ICAM-1 mRNA表达。
64. In contrast with the control, there were many island - like survival myocardium in the infarct aea of the experimental group.
实验组梗塞区内,可见许多呈岛状存活的心肌组织,对照组则极为少见。
65. After 2 hours reperfusion, infarct size and risk region were measured by postmortem dual dyes with triphenyl tetrazolium chloride (TTC) and Evans blue.
家兔处死后以Evans蓝及氯化三苯基四氮唑 (TTC)双重染色确定缺血及梗死心肌范围 ,心肌梗死范围以梗死心肌占危险区心肌重量百分比 (MI/RISK)表示。
66. NS or BM-MNCs was infused into the infarct related artery through the guiding catheter after PCI in control group and trial group respectively.
对照组与试验组在PCI术结束后分别通过大腔导管于梗塞相关血管注入NS与BM-MNCs。
67. In the future VEGF could be gene therapy to decrease the infarct volume and improve the blood flow of the infarct penumbra area.
对神经元也有保护作用，也向我们提供未来一种新的治疗方法，是否可以利用VEGF的基因治疗，减少脑梗塞的面积，改善缺血周围区的血液供应等。
68. Hemorrhagic infarct of small intestine
小肠出血性梗死
69. In io studies in the mouse brain demonstrated that the intraperitoneal administration of leptin, 2 to 8 mg/kg, dose-dependently reduced infarct olume induced by middle cerebral artery occlusion.
小鼠大脑体内研究显示腹腔注射来普汀2-8mg/kg可以剂量依赖性的减少大脑中动脉闭塞所导致的梗塞区域。
70. BACKGROUNDSome cases of acute myocarditi s had elevation of ST segment in the electrocardiogram (ECG), and therefore it was similar with acute myocardial infarct(AMI) and was easily misdiagnosed.
少数急性心肌炎患者的心电图ST段弓背向上抬高,酷似急性心肌梗死表现,易误诊。
71. The main imaging findings of local SLEE which is bad prognosis were large patches cerebral infarct, cerebral hemorrhage, encephalitis.
局灶型的影像学表现多为大片脑梗塞、脑出血、脑炎样改变，其预后较差。
72. The incidence of enhancement region detected in the delayed viability sequence images in the infarct myocardia of 3-days pigs, 3-weeks pigs and 17 patients were respectively 7%, 50% and 88.2%.
心肌活力分析示，左心室心肌内存在不同范围的强化灶，发现率为7.0%、50.0%和88.2%。
73. In the patients with acute inferior myocardial infarction,the presence of ST segment depression in lead aVR was associated with a larger infarct size and ominous progrosis.
急性下壁心肌梗死伴aVR导联ST段压低者梗死面积大,预后不良。
74. This is an acute renal infarction.Note the wedge shape of this zone of coagulative necrosis resulting from loss of blood supply with resultant tissue ischemia that produces the pale infarct.
急性肾梗死，属凝固性坏死，坏死区域呈楔形，是由于组织血供减少引起缺血，终而产生灰白的梗死区。
75. An acute cerebral infarct is seen here. Such infarcts are typically the result of arterial thrombosis or embolism.
急性脑梗死，这种梗死是动脉血栓形成或栓塞的典型表现。
76. To carry out thrombolysis treatment for acute advancement-type cerebral infarction patients could deflate infarct area obviously,abate brain edema,improve nerves function.
急性进展型脑梗死患者于6~24h内采用有效的溶栓治疗,能显著缩小梗死区,减轻脑水肿,改善神经功能。
77. acute non ST segment elevation myocardial infarct
急性非ST段抬高心肌梗死
78. Our recent experiments in pigs have confirmed what we saw in rats: even without cells, the alginate scaffold alone seems to prevent a new infarct from expanding and the ventricle wall from remodeling.
我们最近利用猪只实验再度证实我们在大鼠见到的现象：即使没有植入细胞，褐藻酸盐支架本身似乎就可以防止新梗塞部位继续扩大，以及避免心室的重塑。
79. We believe that growth of new blood vessels in the area of the infarct also contributed greatly to slowing down the tissue deterioration.
我们相信在梗塞区域内新血管的生长，也是大幅减缓恶化的原因。
80. Therefore an embolic infarct has an interface between the affected tissue and surrounding viable normal tissue.
所以脑梗死在累及的组织和周围的正常组织之间有个界限。
