infarcted

infarcted

1. Both MCE and TTC showed that the mass of infarct myocardium of IP group was less than the IR group.

3D MCE及TTC染色结果均显示IP组心肌受损范围小于IR组。

2. The larger the infarct size of CI patient is,the lower the PLT is,the higher the MPV、PDW and Fn is.

4.脑梗塞患者梗塞面积越大，PLT越低，MPV、PDW及血浆Fn越高。

3. CT scan was detected in 50 cases with VD and 141 infarct lesions were discovered (left 79 and right 62).

50例VD患者行CT检查 ,检出141个梗塞灶 ,左侧79个 ,右侧62个。

4. CTA scanning revealed blood vessels convulsion, constriction and obstruction in infarct area.

CTA显示梗塞区血管痉挛、收缩、中断 ,显示出血区造影剂外漏、局部呈无血管区。

5. In focal cerebral ischemia, pretreatment with EDT 2.5, 5, and 10 mg/kg and nimodipine 2 mg/kg for 5 d effectively improved the abnormal neurological symptoms and reduced the infarct rate.

EDT 2.5、5和10mg/kg及尼莫地平2mg/kg灌胃5d显著改善局灶性脑缺血小鼠的神经运动功能,缩小脑梗塞范围。

6. There was significant more infarct in the placenta of ICP patients.

ICP患者胎盘发生梗死率明显高于正常妊娠组;

7. The ratio of RCA to the total number of infarct related arteries (IRA) was much higher than that of the left circumflex artery (LCX) (86.1% versus 13.9%).

IRA的分布也是 RCA多于左旋支 (L CX) ,分别占 86 .1%和 13.9%。

8. RT-3DE can provide a new quantirative approach to identify stunned and infarcted myocardium.

RT-3DE有望为临床定量评价左室局部几何形状提供一项有效手段。

9. Infarct size(IR/AAR)in ATV group were reduced(P<0.01).

TSOD升高、MDA降低(P<0.01)。

10. Some organs such as bowel with anastomosing blood supplies, or liver with a dual blood suppy, are hard to infarct.

一些器官如肠侧支循环较多，或者如肝有双重血液供应，很难发生梗死。

11. Compared with I/R,the myocardial swelling,interstitial hemorrhage and inflammatory cell infiltrate were decreased,the myocardial infarct size was reduced(P<0.01) after CIP and RIP.

与I/R组比较,CIP和RIP均能明显改善心肌损害的形态学改变,降低心肌细胞的肿胀、减少间质出血和炎性细胞的浸润;

12. Haemorrhagic transformation of infarct and severe adverse events were similar in both groups.

两组中梗塞转化为出血及出现严重不良事件的情况类似。

13. Haemorrhagic transformation of infarct and seere aderse eents were similar in both groups.

两组中梗塞转化为出血及出现严重不良反应（应该是＂事件＂，而不是＂反应＂）的情况类似。

14. Conclusion Nicergoline was an effective and safe drug in treatment of multi infarct dementia.

两组均无明显药物相关的不良反应。

15. Volume of infarct focus of rats in the two groups.

两组大鼠梗死灶体积。

16. Hindman N B, Schocken D D, Windmann M, et al. Evaluation of a QRS scoring system for estimating myocardial infarct size[J]. Am J Cardiol,1985,55:1485.

中华心血管病杂志编委会.急性心肌梗死溶栓疗法参考方案[J].中华心血管病杂志,1991,19:37.

17. The net protective of adenosine in invivo models are the reduction of the infarct size and reduce cardiovascular mortality.

临床研究发现腺苷可以减小心肌梗死范围，降低心血管事件死亡率。

18. MAIN OUTCOME MEASURES: Incidence of animal model and infarct volume.

主要观察指标:动物模型出现率;梗死体积。

19. Two of these patients underwent laparotomy, with one surior and one mortality secondary to infarcted small bowel.

之中的两个病人进行了剖腹手术，一个存活，另一个病人死于小肠血管栓塞。

20. Subsequent imaging with MR revealed extension of the infarct into the left posterior cerebral artery territory.

之后的磁共振显示梗塞的范围以扩大到左侧大脑后动脉区域。

21. Behavior disorders were observed at 3, 6, 12h after the operation. Willis circle, infarct volume and histopathologic changes were examined 12 hours after operation.

于手术后 3、6、12h观察行为障碍并评分 ,手术后 12h取脑观察Willis环、脑梗死体积及病理改变。

22. Score 1.5 as criterion,incidence of HF and CS in patient whose QRS score increased over 1.5 was higher than those of under 1.5.Conclusion Patient with RI had larger infarct size than patient with IE.

以 QRS记分增加值 1.5分为界 ,与 IE和 RI发生前比较 QRS记分增加值超过 1.5分者心衰及心脏性休克的发生率明显多于未超过 1.5分者。 结论 RI患者的梗死面积大于 IE患者。

23. Infarcted sizes in heart were determined by dual staining with Evans-blue and TTC.

以Evans蓝和TTC双重染色方法测量心肌梗死面积。

24. Howeer, it is encouraging that cellular therapies, een with current limitations, can significantly improe function of the infarcted heart.

但尽管有现有的种种局限，细胞治疗仍可明显改善梗死心肌心功能，这又令人振奋。

25. Cerebral infarct, intracranial hematoma and edema are deadly diseases, which can cause high deformity and death rate.

但是,目前还缺乏一种无创的辅助诊断技术与设备,用于对颅内血肿水肿进行早期、实时的监测。

26. All the subjects met the criteria for multi infarct dementia.

入组的病例均符合多发性梗死性痴呆的诊断标准。

27. The mechanisms contributed to ischemic brain injury involve excitotoxicity,calcium overload,free radical reaction,depolarization surrounding infarct,inflammation and apoptosis.

兴奋毒性、钙超载、自由基反应、梗死周围除极、炎症和凋亡是造成缺血性脑损伤的中心环节。

28. One of these unanticipated benefits is improement of the infarct's passie mechanical properties and subsequent amelioration of entricular remodeling (23).

其中一个获益原因可能是梗死区心肌被动机械性能的改善，并由此导致的心室重构的改善。

29. So apoptosis may contribute to the final infarct size.

凋亡可能决定了最终梗死体积。

30. Hemorrhagic infarction of the primary disease is cerebral infarction, infarct artery after recanalization, but also hemorrhage.

出血性脑梗塞的原发病是脑梗塞，梗塞的动脉血管再通后，又合并出血。

31. Autologous bone marrow MNCs were injected via the epicardium into the infarcted area in the latter two groups at 1 and 2 weeks after the infarction, respectively.

分为3组：空白对照组、心肌梗死1周后处理组及心肌梗死2周后处理组，处理组经心外膜向心肌注射自体骨髓单核细胞。

32. Initial work-up included a CT demonstrating a medial left occipital infarct involving the left side of the splenium of the corpus callosum.

初始检查CT示左枕叶及中度梗塞并累及左侧胼胝体压部。

33. Here is a cerebral infarct from an arterial embolus, which often leads to a hemorrhagic appearance.

动脉栓子引起的脑梗死,常导致出血性外观。

34. The ultrastructure of myocardium cell was slightly destroyed and the infarct size was significantly smaller in the cap-feed group than the normally feed rabbits and CF rabbits.

卡托普利加胆固醇组心肌细胞超微结构结构破坏及心肌梗死面积明显小于对照组和胆固醇组。

35. Significant swelling in the infarcted area produces obvious shift of the midline by day 5.This corresponded clinically to a diminished level of arousal, which resolved after 2 days.

发病后第5天由于梗塞部位的显著肿胀导致脑中线的偏移，临床上相应表现为意识水平的减低，这个症状2天后可解除。

36. He was also receiving concomitant carvedilol and an ECG showed only an inferior wall infarct without arrhythmia.

另外，他还同时服用卡维地洛，心电图仅显示下壁心梗，没有心律不齐。

37. The subacute (intermediate) infarct seen here at the right shows edema with obscured structural outlines and swelling that shifts the midline to the left.

右边是亚急性梗死(中速)，出现明显水肿，伴组织结构轮廓不清和肿胀，正中线推向左侧。

38. Right frontal encephalomalacia The frontal pole is largely replaced with the high signal of cerebrospinal fluid left after liquifaction necrosis in this old infarct.

右额叶脑软化额极大部分被旧梗塞灶液化坏死后留下的高信号脑脊液替代。

39. Peripheral facial palsy, deafness, tinnitus, and trigeminal sensory deficit are common presentations.Vertigo as the only clinical feature of AICA infarct is rare.

周边颜面神经麻痹、耳聋、耳鸣及三叉感觉异常为其常见的病徵，但只以晕眩症表现的前下下小脑动脉梗塞更是罕见。

40. Instead, noncontractile fibrous cells gradually replace an infarct's dead myocytes.Healthy myocytes adjacent to the infarct may also die, causing the infarct to expand further.

因此到最后，反而是由不具收缩能力的纤维细胞逐渐取代梗塞坏死的心肌细胞，梗塞区域周围的健康心肌细胞也跟著死亡，导致梗塞区域范围扩大。

41. Thus,DPTI realized a valid method to assess the area of acute infarct myocardium of the left ventricle.

因此可将DPTI作为定量分析急性心肌梗塞面积的可靠方法

42. Figure 1. The correlation between infarct size and risk region of I/R and PC groups in rats.

图1.缺血/再灌注组和缺血预处理组大鼠心肌梗死范围与缺血区域的关系.

43. Note the wedge shape of this zone of coagulative necrosis resulting from loss of blood supply with resultant tissue ischemia that produces the pale infarct.

图示凝固性坏死的区域呈楔形是由于该区域血液供应减少而使组织最终发展为贫血性梗死。

44. The infarction seen here has punctate hemorrhages. This infarct was caused by an embolus.

图示梗死为小斑点状出血，梗死由栓子引起。

45. The small intestine is infarcted.

图示：小肠梗死。

46. To gain the effective myocardial reperfusion in the early stage of AMI, the complete and persistent repatency of infarcted related artery (IRA) is the key management to protect ventricular function and reverse VA formation.

在AMI早期阶段得到有效的心肌再灌注，充分持久的开通梗死相关动脉（infarct related artery, IRA）是保护心室功能、逆转VA的形成、改善临床预后的关键措施。

47. The infarct also has high signal on the T2- weighted images. This slice also demonstrates the ribbon-like geometry of the lesion.

在T2加权像上，梗塞灶也呈高信号。此层面损害区域同样呈带状。

48. On the next lower slice, extension of the infarct into white matter is seen.

在下一页更低的层面,可以看到延伸至白质的梗塞灶。

49. Within infarcted hearts in the adult, intramyocardial delivery of iPS yielded progeny that properly engrafted without disrupting cytoarchitecture in immunocompetent recipients.

在成人梗塞心肌中，心肌内进入的iPS产生了正确嫁接的后代，而未对免疫受体细胞结构建造产生影响。

50. The fixed tissue piece were aparted under anatomy scope and then weighted. IS,AAR,IS/AAR% and IS/LV% were observed to assess infarct size.

在解剖显微镜下分离IS、AAR ,分别称重 ,观察IS、AAR、IS/AAR %及IS/LV %以评价缺血范围。

51. A bilateral haemorrhagic infarct was observed in affecting the basal ganglia and thalamus.

基底节和丘脑等处可见双侧出血性梗死。

52. Improving the tolerability of mycardium to hypoxia and act against cardiac infarct like propanolol.

增强心肌耐缺氧能力，有与心得安相似的抗心肌梗塞作用。

53. This is an intermediate to remote infarct in the distribution of the middle cerebral artery.

大脑中动脉供血区域的陈旧性梗死。

54. In the hypothermy environment, obvious increment of human s blood viscosity was observed, which may induce the cerebral infarction, myocardial infarct or coronary heart disease.

太阳活动不仅与人间鼠疫及流行性脑脊髓膜炎两类传染病年发病率间存在着较为密切的相关,而且还与肿瘤的发生和婴儿出生率和死亡率的增加、克山病的发生有关。

55. If the blood supply is not quickly restored, the bowel will infarct.

如果血液供应没有迅速恢复，肠就会梗死。

56. Kahn JC,Guerel P,Menier R,et al.Prognostic value of enzymatic(CPK) estimation of infarct size.

姚竞.急性心肌梗死早期心电图指标与梗死面积的关系.实用心电学杂志,2002；

57. In contrast with the control, there were many island - like survival myocardium in the infarct aea of the experimental group.

实验组梗塞区内,可见许多呈岛状存活的心肌组织,对照组则极为少见。

58. After 2 hours reperfusion, infarct size and risk region were measured by postmortem dual dyes with triphenyl tetrazolium chloride (TTC) and Evans blue.

家兔处死后以Evans蓝及氯化三苯基四氮唑 (TTC)双重染色确定缺血及梗死心肌范围 ,心肌梗死范围以梗死心肌占危险区心肌重量百分比 (MI/RISK)表示。

59. NS or BM-MNCs was infused into the infarct related artery through the guiding catheter after PCI in control group and trial group respectively.

对照组与试验组在PCI术结束后分别通过大腔导管于梗塞相关血管注入NS与BM-MNCs。

60. In the future VEGF could be gene therapy to decrease the infarct volume and improve the blood flow of the infarct penumbra area.

对神经元也有保护作用，也向我们提供未来一种新的治疗方法，是否可以利用VEGF的基因治疗，减少脑梗塞的面积，改善缺血周围区的血液供应等。

61. The main imaging findings of local SLEE which is bad prognosis were large patches cerebral infarct, cerebral hemorrhage, encephalitis.

局灶型的影像学表现多为大片脑梗塞、脑出血、脑炎样改变，其预后较差。

62. Can stem cells regenerate infarcted myocardiocytes?

干细胞能再生梗死心肌吗?

63. Effects of Transmyocardial Revascularization with Frequency-double Nd:YAG Laser on Myocardial Blood Flow of Acute Infarcted Myocardium in Canine[J].

引用该论文 蒋桔泉,曹林生,曾秋棠,聂绍平,曹国祥,刘小青,李正佳,朱长虹.

64. In the patients with acute inferior myocardial infarction,the presence of ST segment depression in lead aVR was associated with a larger infarct size and ominous progrosis.

急性下壁心肌梗死伴aVR导联ST段压低者梗死面积大,预后不良。

65. The acutely edematous infarcted tissue may produce a mass effect.

急性水肿性梗死组织可形成团块效应。

66. An acute cerebral infarct is seen here. Such infarcts are typically the result of arterial thrombosis or embolism.

急性脑梗死，这种梗死是动脉血栓形成或栓塞的典型表现。

67. To carry out thrombolysis treatment for acute advancement-type cerebral infarction patients could deflate infarct area obviously,abate brain edema,improve nerves function.

急性进展型脑梗死患者于6~24h内采用有效的溶栓治疗,能显著缩小梗死区,减轻脑水肿,改善神经功能。

68. Our recent experiments in pigs have confirmed what we saw in rats: even without cells, the alginate scaffold alone seems to prevent a new infarct from expanding and the ventricle wall from remodeling.

我们最近利用猪只实验再度证实我们在大鼠见到的现象：即使没有植入细胞，褐藻酸盐支架本身似乎就可以防止新梗塞部位继续扩大，以及避免心室的重塑。

69. We believe that growth of new blood vessels in the area of the infarct also contributed greatly to slowing down the tissue deterioration.

我们相信在梗塞区域内新血管的生长，也是大幅减缓恶化的原因。

70. We also use granulocyte colony-stimulating factor mobilize bone marrow to treat acute myocardial infarction.This method could reduce infarcted area and protect cardiac function.

我们还进一步研究了应用粒细胞集落刺激因子骨髓动员治疗急性心肌梗死，发现这一方法可减少梗死面积，保护心脏功能。

71. Therefore an embolic infarct has an interface between the affected tissue and surrounding viable normal tissue.

所以脑梗死在累及的组织和周围的正常组织之间有个界限。

72. Diffusion-weighted imaging (DWI), which successfully discriminats acute from chronic infarct, is particularly useful in patients with multiple infarcts in more than one territory.

扩散磁振造影因能分辨急性与慢性病灶而成为检查急性多发性脑梗塞的利器。

73. Our purpose is to establish a steady and credible animal model of infarct heart.

摘要介绍了如何建立可靠稳定的心肌梗塞动物模型。

74. Objective To explore the clinical value of changes of depressive emotion and P300 (EP) evoked potentials in patients with lacunar infarct (LI).

摘要目的探讨腔隙性脑梗死(LI)患者的抑郁情绪和P300电位变化及其临床价值。

75. Abstract: Objective： To study the effects of myocardial angiogenesis and molecular mechanisms on infarcted rats after taking Yiqihuoxue prescription.

文章摘要: 目的：探讨益气活血方对心梗后大鼠缺血心肌局部血管新生的影响及其分子学机制。

76. Methods:To forecast the Infarct area by QRS score, LVEF by colour doppler echocardiography and detect SOD and MDA.

方法 :采用QRS记分法预测梗死面积、彩色超声多普勒测定射血分数 (LVEF)及血清超氧化物歧化酶 (SOD)、丙二醛 (MDA)含量检测。

77. Method:We retrospectively analyzed the clinical data and the features of accessory examination of 44 cases of lobar infarct,and the diagnosis was confirmed by CT and MRI.

方法:回顾性分析44例经CT或MRI确诊的脑叶梗塞患者的临床资料及辅助检查特点,对其危险因素、临床表现、诊断、治疗进行分析。

78. Methods 100 ACS patients were divided into unstable angina pectoris(UAP) group(n=64),acute myocardial infarct (AMI) group(n=36)and 50 healthy cases as control group.

方法ACS患者100例分为不稳定型心绞痛(UAP)组64例和急性心肌梗死(AMI)组36例,正常对照组50例。

79. Methods 20 cases with cerebral infarct were studied with FLAIR sequence in and FSE.

方法对40例脑梗塞病人行快速自旋回波(FSE)序列及FLAIR序列扫描。

80. Methods To study the effects of electrocardiograph (ECG), myocardial enzymes and myocardial infarct scope in experimental rats by ligating the coronary arteries.

方法通过大鼠冠脉结扎致心肌缺血模型,观察通心活脉灵对大鼠心电图、心肌酶及梗死面积的影响。